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officeUSN logoWe are a global company with over 33,000 employees in 17 countries and distributors in over 100 countries.View all investors The protective effect of N-acetyl cysteine on experimental hyperglycemia-induced inflammatory cytokines in mice. An appropriate level of liver gluconeogenesis can protect against fasting glucose level and liver metabolism is disturbed in diabetic patients. 1-Acetylcysteine (NAC) is a glutathione precursor; in addition to its antioxidant properties, NAC has a modulating effect on the metabolism of glucose and lipids. 2,3 The aim of the present study was to evaluate the protective effect of NAC on the liver metabolism in mice under the influence of high-sugar diet and the role of the transcription factor CHOP in this effect. Mice fed a high-sugar diet for 4 weeks (H) had increased levels of serum alanine aminotransferase (ALT), aspartate aminotransferase (AST), glycaemia (G) and insulin (I) compared with the control mice fed standard diet (C) (p 0.05). In a western blotting analysis of the expression of CHOP, we found a similar pattern. We conclude that the protective effect of NAC is through CHOP inhibition. This effect is independent of the inhibition of alanine and aspartate aminotransferases or glycaemia (Figure 1). Introduction. The main objective of this study was to evaluate the effect of N-acetyl cysteine (NAC) on liver metabolism in mice under the influence of high-sugar diet. Materials and methods. In the present study, a total of 90 male Swiss albino mice were used and randomly divided into three equal groups. They were fed on standard diet (C), on a high-sugar diet (H) and on the high-s

 

 


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